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          Thursday, November 23, 2017

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Effect of Brahmi on nitric
oxide-related toxicity

There is growing evidence that high concentrations of nitric oxide (NO), generated by activated astrocytes, might be involved in a variety of neurodegenerative diseases, such as Alzheimer's disease, ischemia and epilepsy. It has recently been suggested that glial cells may produce NO under superoxide radical stimulation by enzyme-independent mechanism. This suggests that also natural antioxidants may have therapeutical relevance in neurodegenerative diseases.

Studies of Bhattacharya et al. have evidenced that Brahmi is able to reduce the memory-dysfunction in rat models of Alzheimer's disease, but the molecular mechanisms of this action are yet to be determined.

In a study at Department of Biological Chemistry, Medical Chemistry and Molecular Biology, University of Catania, Catania, Italy, the researchers examined the effect of a methanolic extract of Brahmi on toxicity induced by the nitric oxide donor, S-nitroso-N-acetyl-penicillamine (SNAP), in culture of purified rat astrocytes.

The results indicate that after 18 h of treatment, SNAP induced an increase in the production of reactive species, but did not induce the rupture of cellular membrane. Conversely, this NO donor induced a fragmentation of genomic DNA compared to control astrocytes. The extract of Brahmi inhibited the formation of reactive species and DNA damage in a dose dependent manner; an observation that supports the traditional use of Brahmi and indicates that this medicinal plant has a therapeutic potential in treatment or prevention of neurological diseases.

Brahmi extract reduces morphine-
induced withdrawal symptoms


Evaluation of antistress effect of Brahmi

In vitro effect of Brahmi on anti-Helicobacter pylori activity

Free radical scavenging capacity of Brahmi on DNA damage

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